HELP! Мне говорят - ты храпишь, как сапожник...

Специалисты! Сочувствующие! Психологи!
Помогите, я в отчаянии. Я храплю с детства, громко, в любом положении. Правда, видимо, не всю ночь напролет, иначе было бы удивительно, как мне удалось прожить замужем 15 лет.
Меня лично это не беспокоит, я сплю крепко, вижу сны, отдыхаю во сне. Но только не сейчас, не в этом историческом промежутке времени.
Сейчас я пытаюсь построить новую счастливую жизнь с новым избранником, и все великолепно, если бы не храп. А мой любимый мужчина много работает, и ему так хочется нормально выспаться.
А тут - жуткий храп спящей на его плече любимой женщины. Вот вам и социальный аспект.
А я стараюсь не спать и прислушиваться к себе, и как только всхрапываю, тут же просыпаюсь, слышу, что он не спит, расстраиваюсь, лежу тихо, снова начинаю засыпать, опять всхрапываю... и т.д.
Утром он встает с таким видом... а я с таким чувством... Мне нечем его утешить, а ему - меня упрекнуть. Нам обоим плохо.
Мне страшно ложиться спать. Ему не хочется оставаться у меня. Спать в разных комнатах? А какой при этом смысл жить вместе? Мы и так-то видимся только от заката до рассвета...

Скажите, какие тут шансы? Что можно сделать? Что сказать?
Я в отчаянии. Помогите, ответьте.
Тут на консультацию записалась было, регистраторша ошиблась, сказала, приходите в 10, а врач принимал с 11. А каждая новая совместно проводимая ночь может быть последней. А врач-то раз в неделю...

[buzunov]

Уважаемая Alison!

Медрегистратор ошиблась вдвойне, так как принимал с 10.00 и этом момент находился в клинике. Даже, если не ошибаюсь, мимо Вас прошел как раз когда Вы о чем-то с ней бесседовали.

Так что придется предпринять еще одну попытку.

Что касается выбора тактики лечения, то она зависит от причин и тяжести храпа. Более подробно об этом можно почитать на сайте [Ссылки доступны только зарегистрированным пользователям ]


Не исключено, что в Вашей ситуации (большая длительность храпа) для уточнения тяжести может потребоваться полисомнографическое исследование.

Более подробную информацию Вы можете получить в нашей лаборатории сна по тел. 418-69-07.

[Doktor_dev]

Почти та же ситуация. Храпел и храпел, никому не мешало. Немного избыточная масса тела, киллограм 10 точнее. Нервная работа, ночные дежурства. Встретил женщину, любовь, собираюсь жениться, НО! Мой храп не даёт ей спать. Мучаюсь, пытаясь не засыпать, с утра сплю на работе. Ловлю косые взгляды коллег. Доходит до маразма, полночи я сплю в другой комнате . Что делать? Поспрашивал у коллег, Лор ответил невнятно, типа носовая перегородка искривлена, можно подправить, а можно и нет, гарантии нет.
Такие вот дела.

[Dr.Vad]

Вот такие способы борьбы с храпом /нарушением дыхания во время сна существуют:

J R Soc Med 2003;96:343-344

Treatment options for snoring and sleep apnoea

B Kotecha MPhil FRCS J M Shneerson DM FRCP
Royal National Throat, Nose and Ear Hospital, Gray's Inn Road, London W1X 9DA
Papworth Hospital, Papworth Everard, Cambridge CB3 8RE, UK

Snoring is due to turbulent flow of air through the upper airway, and can arise from transient obstruction at several levels. This multi-segmental obstruction occurs in different regions of the pharynx in varying proportions in different individuals. In some patients it is predominantly palatal and nasopharyngeal, whereas in others it may be retroglossal or hypopharyngeal.

Obstruction occurs because of a failure of the upper airway dilator muscles to stabilize the airway during sleep. Their innervation is complex and subject to reflex control. The trauma to the upper airway tissues during snoring and sleep apnoeas damages either muscle fibres or peripheral nerve fibres and this impairs the stabilizing action of the muscles and increases the tendency of the airway to obstruct. The obstruction is compounded by oedema due to the vibration injury during snoring, and these factors, quite independently of the ageing process and obesity, may account for the progression of snoring into sleep apnoeas in some individuals.

Investigative techniques have been developed to predict which patients will benefit from oral appliances or surgery rather than nasal continuous positive air pressure (CPAP), which although effective4 is often difficult to use properly. Nasopharyngoscopy is performed routinely by otolaryngologists to evaluate the upper airway, both during wakefulness and during sleep. 'Sleep' nasendoscopy allows dynamic examination of the pharynx during drug-induced sedation which simulates sleep. The level at which the airway obstructs can be visualized and site-specific treatment can then be provided. Acoustic analysis of snoring has indicated that frequencies around 20 Hz are associated with palatal fluttering whereas obstruction at other sites gives a more diffuse frequency range. A limitation is the considerable variation in the frequencies from night to night, and the technique cannot be used to predict which individuals have sleep apnoeas rather than simple snoring. Conventional sleep studies are required for this, but developments in software to analyse the snoring duration, loudness, periodicity and frequency may become of clinical value.

Mandibular advancement devices or splints fit over the anterior teeth like a gum shield and hold the mandible and tongue forwards. Many are thermoplastic and can be moulded by the patient; others are individually constructed, by dental surgeons for instance. Various designs are in use but there are few data to indicate which is most effective. The current indications for these devices are snoring when surgery is not advisable (e.g. when the snoring occurs at the level of the tongue base) and mild to moderate sleep apnoeas, particularly in thin people and those in whom the apnoeas occur predominantly in the supine position. They also have a role in individuals with more severe sleep apnoeas who are intolerant of nasal CPAP treatment. Mandibular advancement devices may affect dental occlusion and the temporomandibular joints.

Surgical treatments can be targeted at different anatomical regions in the upper airway. Nasal surgery such as septoplasty, functional endoscopic sinus surgery and turbinate reduction may be effective if there is nasal obstruction, and nasal abnormalities should be looked for in those who develop nasal symptoms during CPAP treatment. Mandibular, maxillary and hyoid surgery are only occasionally performed and tracheostomy is seldom required, because other treatment options are available. Although palatal surgery has been popular since the 1980s, surgical uvulopalatopharyngoplasty carries a high morbidity which may be avoidable by new laser techniques. The short-term results of these are good in selected patients10 although late recurrence of symptoms has been reported. Radiofrequency thermal ablation with somnoplasty12 and with the Celon device are less painful than a laser palatoplasty and merit further evaluation.

The prevalence of sleep apnoeas in children is around 3%, with the highest frequency between the ages of 2 and 5 years. Hypertrophy of tonsils and adenoids is the commonest cause, but craniofacial abnormalities such as arise in Pierre Robin and Down's syndromes may be responsible. Compliance with nasal CPAP may be difficult to achieve14 and surgical intervention is effective in carefully selected cases. Craniofacial surgery should be confined to specialist centres.

Although a wide range of treatments is now available for snoring and sleep apnoeas, objective evidence of outcome benefits is sparse. The impact of snoring and sleep apnoeas on the quality of life of the patient and partner needs to be evaluated and the short and long-term benefits from surgical and non-surgical treatments compared. Newer psychometric techniques are likely to be useful and underline the importance of a multidisciplinary approach to the management of snoring and sleep apnoeas.

Кроме, того показан повышенный риск СС патологии у таких пациентов вероятно вследствие нарушения толерантности к глюкозе:

Respir Physiol Neurobiol. 2003 Jul 16; 136(2-3): 167-78.

Sleep-disordered breathing, glucose intolerance, and insulin resistance.

Punjabi NM, Ahmed MM, Polotsky VY, Beamer BA, O'Donnell CP.

Division of Pulmonary and Critical Care Medicine, Johns Hopkins Asthma and Allergy Center, 5501 Hopkins Bayview Circle, Baltimore, MD 21224, USA. [Ссылки доступны только зарегистрированным пользователям ]

Sleep-disordered breathing (SDB) is a common condition with prevalence estimates of 2-4% in the general population. Epidemiological data suggest that SDB is an independent risk factor for cardiovascular disease. Glucose intolerance and insulin resistance are also well-recognized risk factors for the development of cardiovascular disease. A number of recent clinic-based studies suggest that, independent of obesity, SDB may adversely affect glucose tolerance and insulin sensitivity. The purpose of this study was to systematically review the evidence for the link between SDB, glucose intolerance, and insulin resistance. A MEDLINE search for SDB and metabolic disorders was performed and 24 articles that met the inclusion criteria were identified. Population-based studies indicate that habitual snoring is independently associated with glucose intolerance and insulin resistance. Studies that have used objective measures of SDB (e.g. polysomnography) provide further support for an independent link between SDB, glucose intolerance, and insulin resistance. However, studies on the treatment of SDB with continuous positive airway pressure (CPAP) have yielded inconsistent results and overall do not reveal an improvement in the metabolic disturbance after treatment. Although population-based prospective data on the metabolic implications of SDB are still lacking, current data point to an independent association between SDB and impaired glucose homeostasis.