нормохромной анемия может быть из-за сочетания дефицита железа и фолиевой, но все равно может ответить на лечение железом;
гемолиз должен быть существенным, чтобы вызвать гипер-фосфатемию, вот осн. причины
Renal failure is the most common cause of hyperphosphatemia. A glomerular filtration rate of less than 30 mL/min significantly reduces the filtration of inorganic phosphate, increasing its serum level.
Other less common causes include a high intake of phosphorus or increased renal reabsorption.
High intake of phosphate can result due to excessive use of phosphate-containing laxatives or enemas, and vitamin D intoxication. Vitamin D increases intestinal phosphate absorption.
Hyperphosphatemia can also be due to genetic causes. Several genetic deficiencies can lead to hypoparathyroidism, pseudohypoparathyroidism, and decreased FGF-23 activity.
Pseudohyperphosphatemia is a laboratory artifact sometimes seen in patients with hyperglobulinemia, hyperlipidemia, and hyperbilirubinemia. This artifact is due to interference in phosphate assay.
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