[Dr.Anisimova]

- в т.ч. последний нормер Индийского журнала, подборка по витилиго:
A single nucleotide polymorphism rs9468925 of MHC region is associated with clinical features of generalized vitiligo in Chinese Han population
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Vitiligo has been found to be associated with different HLA antigens in different ethnic groups. In our previous genome-wide association study (GWAS), we identified independent association signal of rs9468925 (P = 2.21 ? 10?33, OR = 0.74) within HLA-C-HLA-B region.
Conclusions Allele G of rs9468925 on HLA-C-HLA-B may be associated with a higher risk of vitiligo. Our study showed a significant genotypic variation between patients with age of onset ?20*years and age of onset >20*years. Obvious clinical differences of generalized vitiligo related to genotypic variation found in the Chinese Han population were confirmed in this study.
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Basic fibroblast growth factor and tumour necrosis factor alpha in vitiligo and other hypopigmented disorders: suggestive possible therapeutic targets
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Conclusion The study’s findings demonstrate that the studied hypopigmented (vitiligo, hypopigmented MF, hypopigmented TV) disorders show similar changes in their cutaneous microenvironment with increased TNF- and decreased bFGF mRNA expression. This cytokine microenvironment change may be implicated in the pigment loss and hence these cytokines may have future therapeutic implications.

Targeting Skin: Vitiligo and Autoimmunity[Ссылки доступны только зарегистрированным пользователям ]
Survey of dermatologists' phototherapy practices for vitiligo
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Segmental vitiligo: A randomized controlled trial to evaluate efficacy and safety of 0.1% tacrolimus ointment vs 0.05% fluticasone propionate cream
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Our study suggests that both topical tacrolimus and fluticasone have a limited efficacy in segmental vitiligo in terms of number of patients showing repigmentation and the percentage area showing repigmentation. The side effects are comparable, transient and acceptable. However, the use of either topical tacrolimus or fluticasone in segmental vitiligo cannot be completely discarded as it may have a role to play in halting further progression of the disease.

The effect of H 1 and H 2 receptor antagonists on melanogenesis[Ссылки доступны только зарегистрированным пользователям ]
Based on these results, [12] two studies emerged; the first explained why vitiligo patients who were on antihistaminic therapy were not responding to conventional vitiligo therapy [13] and the second one showed that pigmentation is synchronized by a pH change during melanosome maturation and that the switch from pH 5.0 to 6.8 seems to depend on the proton pump p-protein in the melanosome membrane. [14]

Non-cultured epidermal suspension in vitiligo: From laboratory to clinic[Ссылки доступны только зарегистрированным пользователям ]
The non-cultured epidermal suspension techniques are safe, effective, and simpler than the other methods involving cell culturing and requiring a laboratory set-up. Selection of patients is crucial for the success of the outcome.

Depigmentation therapies in vitiligo
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To conclude, depigmentation therapies are the last resort for extensive vitiligo cases. These therapies are evolving and new therapies have come in vogue apart from MBEH and 4 MP. In future, some experimental products may become mainstay of depigmentation therapies.

Ultraviolet-based therapy for vitiligo: What's new?[Ссылки доступны только зарегистрированным пользователям ]

Childhood vitiligo
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On the pathophysiology of vitiligo: Possible treatment options[Ссылки доступны только зарегистрированным пользователям ]

Melanocytorrhagy and apoptosis in vitiligo: Connecting jigsaw pieces[Ссылки доступны только зарегистрированным пользователям ]

Vitiligo: Emerging paradigms[Ссылки доступны только зарегистрированным пользователям ]

Camouflage for patients with vitiligo
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Measuring the severity of vitiligo
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