[Изображения доступны только зарегистрированным пользователям] Этиологическими не являются, но заболевания провоцируют...
Д-р Зайцев, я специально не искал...
Я готовлю небольшой доклад по другой теме, но попадаются и про озон сообщения.
[Ссылки доступны только зарегистрированным пользователям ] A major source of ozone (O3) is motor vehicle exhaust. O3 is a main constituent of photochemical smog, and its formation requires NOX and reactive organic compounds as precursors and ultraviolet radiation (Gorski & Tarkowski, 1992; Wardlaw, 1993; Beggs & Curson, 1995). It is the most ubiquitous air pollutant in the USA (Koenig, 1995). Although O3 is mainly an outdoor pollutant, it is also present in low concentrations in the indoor environment (Koren, 1995). Many studies have shown that O3 has an aggravating effect in asthmatics and can reduce lung function (Krzyzanowski et al., 1992; Gorski & Tarkowski, 1992; Wardlaw, 1993; Koenig, 1995; Koren & Bromberg, 1995). Unlike SO2 there seems to be no marked difference in acute responses in asthmatics and non-asthmatics (Koenig et al., 1988). Animal models suggest that O3, like other air pollutants, may increase sensitization to allergens (Osebold et al., 1980; Sears et al., 1989). Ambient levels of O3 may also have a synergistic effect with pollen in the causation of allergic rhinitis (Bascom et al., 1990). However, only a few epidemiological investigations have been performed on a population level to evaluate the effect of ambient O3 on asthma and allergies (e.g., Braun-Fahrlander et al., 1997) and further investigations are needed to study the effect of O3 on these disorders under real-life exposure conditions (Magnussen et al., 1998).
Весь документ, кстати, неплохой, но весит больше мега...
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